XB-FEAT-5918262: Difference between revisions
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=kcnj8= | =kcnj8= | ||
This is the community wiki page for the gene ''kcnj8'' please feel free to add any information that is relevant to this gene that is not already captured elsewhere in Xenbase | This is the community wiki page for the gene ''kcnj8'' please feel free to add any information that is relevant to this gene that is not already captured elsewhere in Xenbase. | ||
=nomenclature changes= | |||
08.23.2019 | |||
Human name has changed for Entrez Gene: 3764. From potassium voltage-gated channel subfamily J member 8 to potassium inwardly rectifying channel subfamily J member 8 | |||
=Summary from NCBI for human KCNJ8= | |||
Potassium channels are present in most mammalian cells, where they participate in a wide range of physiologic responses. The protein encoded by this gene is an integral membrane protein and inward-rectifier type potassium channel. The encoded protein, which has a greater tendency to allow potassium to flow into a cell rather than out of a cell, is controlled by G-proteins. Defects in this gene may be a cause of J-wave syndromes and sudden infant death syndrome (SIDS). [provided by RefSeq, May 2012] | |||
Latest revision as of 06:30, 27 August 2019
kcnj8
This is the community wiki page for the gene kcnj8 please feel free to add any information that is relevant to this gene that is not already captured elsewhere in Xenbase.
nomenclature changes
08.23.2019
Human name has changed for Entrez Gene: 3764. From potassium voltage-gated channel subfamily J member 8 to potassium inwardly rectifying channel subfamily J member 8
Summary from NCBI for human KCNJ8
Potassium channels are present in most mammalian cells, where they participate in a wide range of physiologic responses. The protein encoded by this gene is an integral membrane protein and inward-rectifier type potassium channel. The encoded protein, which has a greater tendency to allow potassium to flow into a cell rather than out of a cell, is controlled by G-proteins. Defects in this gene may be a cause of J-wave syndromes and sudden infant death syndrome (SIDS). [provided by RefSeq, May 2012]